Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. On the flip side, our capability to rescue SERT perform in KI midbrain synaptoneurosomes from the inhibition of FAK suggests elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by greater pFAK localization in 5-HT https://miltonf987tff6.blogunok.com/profile